* 背景图:Myocardium with coagulation necrosis, showing strongly eosinophilic anucleate myocardial fibers. Leukocytes in the interstitium are an early reaction to necrotic muscle. Compare with A and with normal fibers in the lower part of the figure. 左下图:Normal myocardium. * An acute cerebral infarct is seen here. * The infarction of bilaterally symmetric dark discolored areas. This is an intermediate to remote infarct in the distribution of the middle cerebral artery. Here is a cerebral infarct from an arterial embolus, which often leads to a hemorrhagic appearance. This intermediate infarct of the frontal lobe shows liquefactive necrosis with formation of cystic spaces as resolution begins. * A lacunar infarct is a cystic space from the resolved liquefactive necrosis. * Resolution of the liquefactive necrosis in a cerebral infarction leads to the formation of a cystic space. * This cerebral infarction demonstrates the presence of many macrophages at the right(liquefactive necrosis). * Hemorrhagic, roughly wedge-shaped pulmonary infarct. * Remote kidney infarct, now replaced by a large fibrotic cortical scar. * Factors affecting fluid balance across capillary walls. Capillary hydrostatic and osmotic forces are normally balanced so that there is no net loss or gain of fluid across the capillary bed. However, increased hydrostatic pressure or diminished plasma osmotic pressure leads to a net accumulation of extravascular fluid (edema). As the interstitial fluid pressure increases, tissue lymphatics remove much of the excess volume, eventually returning it to the circulation via the thoracic duct. If the ability of the lymphatics to drain tissue is exceeded, persistent tissue edema results. * The surface of the brain with cerebral edema demonstrates widened gyri with a flattened surface. * Note the marked dilation of the cerebral ventricles. This is hydrocephalus. 丝虫病 象皮病(elephantiasis) 图1 图3 图2 图4 图5 图6 * Catheter 导管 * The extensive white matter petechial hemorrhage
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